雅思阅读题难度不高,但还是经常有很多考生做不对。今天小编给大家带来了雅思阅读高分技巧之抓住数字找答案,希望能够帮助到大家,下面小编就和大家分享,来欣赏一下吧
【阅读题型讲解】雅思阅读选择题答题技巧
雅思阅读选择题目前在每周一次的考试趋势中,还是相当重要的。如此高频的题目,却又被认为是“特别好定位,总是做不对”,继四大主流题型之后的又一“让人会呼吸的疼”。小站老师将在本文中以真题为例帮大家详细讲解做选择题的方法和步骤。
我们一起来看看雅思专家来分析一道雅思六Test 4 Reading Passage 3的第32题:
Until recently, not much was known about the topic, and little help was available to teachers to deal with bullying. Perhaps as a consequence, schools would often deny the problem. “There is no bullying at this school” has been a common refrain, almost certainly untrue. Fortunately more schools are now saying,” there is not much bully here, but when it occurs we have a clear policy for dealing with it.”
The writer thinks that the declaration “There is no bullying at this school”
A. is no longer true in many schools
B. was not in fact made by many schools
C. reflected the school’s lack of concern
D. reflected a lack of knowledge and resources
第一,明确一点,雅思阅读选择题是顺序原则的题。通常来说,一个段落定位一道题,最多一个段落会出现两道选择题的定位点。
第二,先读题干,通过题干到文中定位,题干中的话加上了引号,说明是引用,发现在文中显而易见,在该段的第三行,很多学生习惯性的看该句的后面,很快遇到了生词refrain,然后再往下看,看到了untrue, 发现和第一个选项很相像,所以毫不犹豫选A。其实选择题里最具有干扰的、最具有迷惑性的就是选项和文中的句子长得相似,多半是错误答案。还有一部分学生是往前看了,看到了schools deny the problem, 学校否认问题,立马产生联想,学校一定是缺乏关心,才否认问题,所以立刻选C, 这样的学生犯了最大的错,就是过度推断,雅思阅读只考察字面意义上的同义转换。其实deny the problems的前面,出现了很重要的逻辑关系词as a consequence, 这一词组后面接的是结果,前面是原因,题干定位在as a consequence的后面,说明这道题是想考察产生这件事的原因,所以答案是这段第一句话的同义改写。
第三,定位到确切的句子,找同义改写not much was known about the topic, and little help was available to teachers to deal with bullying. 这句话中出现了两个否定,一是对欺负不了解,二是得到老师的帮助来处理欺负很少,符合D选项中的否定词lack, knowledge and resources.
雅思考试阅读模拟试题及答案解析
1 There’s a dimmer switch inside the sun that causes its brightness to rise and fall on timescales of around 100,000 years - exactly the same period as between ice ages on Earth. So says a physicist who has created a computer model of our star’s core.
2 Robert Ehrlich of George Mason University in Fairfax, Virginia, modelled the effect of temperature fluctuations in the sun’s interior. According to the standard view, the temperature of the sun’s core is held constant by the opposing pressures of gravity and nuclear fusion. However, Ehrlich believed that slight variations should be possible.
3 He took as his starting point the work of Attila Grandpierre of the Konkoly Observatory of the Hungarian Academy of Sciences. In 2005, Grandpierre and a collaborator, Gábor ágoston, calculated that magnetic fields in the sun’s core could produce small instabilities in the solar plasma. These instabilities would induce localised oscillations in temperature.
4 Ehrlich’s model shows that whilst most of these oscillations cancel each other out, some reinforce one another and become long-lived temperature variations. The favoured frequencies allow the sun’s core temperature to oscillate around its average temperature of 13.6 million kelvin in cycles lasting either 100,000 or 41,000 years. Ehrlich says that random interactions within the sun’s magnetic field could flip the fluctuations from one cycle length to the other.
5 These two timescales are instantly recognisable to anyone familiar with Earth’s ice ages: for the past million years, ice ages have occurred roughly every 100,000 years. Before that, they occurred roughly every 41,000 years.
6 Most scientists believe that the ice ages are the result of subtle changes in Earth’s orbit, known as the Milankovitch cycles. One such cycle describes the way Earth’s orbit gradually changes shape from a circle to a slight ellipse and back again roughly every 100,000 years. The theory says this alters the amount of solar radiation that Earth receives, triggering the ice ages. However, a persistent problem with this theory has been its inability to explain why the ice ages changed frequency a million years ago.
7 "In Milankovitch, there is certainly no good idea why the frequency should change from one to another," says Neil Edwards, a climatologist at the Open University in Milton Keynes, UK. Nor is the transition problem the only one the Milankovitch theory faces. Ehrlich and other critics claim that the temperature variations caused by Milankovitch cycles are simply not big enough to drive ice ages.
8 However, Edwards believes the small changes in solar heating produced by Milankovitch cycles are then amplified by feedback mechanisms on Earth. For example, if sea ice begins to form because of a slight cooling, carbon dioxide that would otherwise have found its way into the atmosphere as part of the carbon cycle is locked into the ice. That weakens the greenhouse effect and Earth grows even colder.
9 According to Edwards, there is no lack of such mechanisms. "If you add their effects together, there is more than enough feedback to make Milankovitch work," he says. "The problem now is identifying which mechanisms are at work." This is why scientists like Edwards are not yet ready to give up on the current theory. "Milankovitch cycles give us ice ages roughly when we observe them to happen. We can calculate where we are in the cycle and compare it with observation," he says. "I can’t see any way of testing [Ehrlich’s] idea to see where we are in the temperature oscillation."
10 Ehrlich concedes this. "If there is a way to test this theory on the sun, I can’t think of one that is practical," he says. That’s because variation over 41,000 to 100,000 years is too gradual to be observed. However, there may be a way to test it in other stars: red dwarfs. Their cores are much smaller than that of the sun, and so Ehrlich believes that the oscillation periods could be short enough to be observed. He has yet to calculate the precise period or the extent of variation in brightness to be expected.
11 Nigel Weiss, a solar physicist at the University of Cambridge, is far from convinced. He describes Ehrlich’s claims as "utterly implausible". Ehrlich counters that Weiss’s opinion is based on the standard solar model, which fails to take into account the magnetic instabilities that cause the temperature fluctuations.
Questions 1-4
Complete each of the following statements with One or Two names of the scientists from the box below.
Write the appropriate letters A-E in boxes 1-4 on your answer sheet.
A. Attila Grandpierre
B. Gábor ágoston
C. Neil Edwards
D. Nigel Weiss
E. Robert Ehrlich
1. ...claims there a dimmer switch inside the sun that causes its brightness to rise and fall in periods as long as those between ice ages on Earth.
2. ...calculated that the internal solar magnetic fields could produce instabilities in the solar plasma.
3. ...holds that Milankovitch cycles can induce changes in solar heating on Earth and the changes are amplified on Earth.
4. ...doesn’t believe in Ehrlich’s viewpoints at all.
Questions 5-9
Do the following statements agree with the information given in the reading passage?
In boxes 5-9 on your answer sheet write
TRUE if the statement is true according to the passage
FALSE if the statement is false according to the passage
NOT GIVEN if the information is not given in the passage
5. The ice ages changed frequency from 100,000 to 41,000 years a million years ago.
6. The sole problem that the Milankovitch theory can not solve is to explain why the ice age frequency should shift from one to another.
7. Carbon dioxide can be locked artificially into sea ice to eliminate the greenhouse effect.
8. Some scientists are not ready to give up the Milankovitch theory though they haven’t figured out which mechanisms amplify the changes in solar heating.
9. Both Edwards and Ehrlich believe that there is no practical way to test when the solar temperature oscillation begins and when ends.
Questions 10-14
Complete the notes below.
Choose one suitable word from the Reading Passage above for each answer.
Write your answers in boxes 10-14 on your answer sheet.
The standard view assumes that the opposing pressures of gravity and nuclear fusions hold the temperature ...10...in the sun’s interior, but the slight changes in the earth’s ...11... alter the temperature on the earth and cause ice ages every 100,000 years. A British scientist, however, challenges this view by claiming that the internal solar magnetic ...12... can induce the temperature oscillations in the sun’s interior. The sun’s core temperature oscillates around its average temperature in ...13... lasting either 100,000 or 41,000 years. And the ...14... interactions within the sun’s magnetic field could flip the fluctuations from one cycle length to the other, which explains why the ice ages changed frequency a million years ago.
Answer keys and explanations:
1. E
See the sentences in paragraph 1(There’s a dimmer switch inside the sun that causes its brightness to rise and fall on timescales of around 100,000 years - exactly the same period as between ice ages on Earth. So says a physicist who has created a computer model of our star’s core.) and para.2 (Robert Ehrlich of George Mason University in Fairfax, Virginia, modelled the effect of temperature fluctuations in the sun’s interior.)
2. A B
See para.3: ?i style=’normal’>Grandpierre and a collaborator, Gábor ágoston, calculated that magnetic fields in the sun’s core could produce small instabilities in the solar plasma.
3. C
See para.8: Edwards believes the small changes in solar heating produced by Milankovitch cycles are then amplified by feedback mechanisms on Earth.
4. D
See para.11: Nigel Weiss, a solar physicist at the University of Cambridge, is far from convinced. He describes Ehrlich’s claims as "utterly implausible".
5. False
See para.5: for the past million years, ice ages have occurred roughly every 100,000 years. Before that, they occurred roughly every 41,000 years.
6. False
See para.7: "In Milankovitch, there is certainly no good idea why the frequency should change from one to another," ... Nor is the transition problem the only one the Milankovitch theory faces.
7. Not Given
See para.8: if sea ice begins to form because of a slight cooling, carbon dioxide?is locked into the ice. That weakens the greenhouse effect. (The passage doesn抰 mention anything about locking Co2 into ice artificially.)
8. True
See para.9: there is no lack of such mechanisms. "If you add their effects together, there is more than enough feedback to make Milankovitch work,"?"The problem now is identifying which mechanisms are at work." This is why scientists like Edwards are not yet ready to give up on the current theory.
9. True
See the sentences in para.9 (According to Edwards, 卙e says. "I can’t see any way of testing [Ehrlich’s] idea to see where we are in the temperature oscillation.") and para.10 (Ehrlich concedes this. "If there is a way to test this theory on the sun, I can’t think of one that is practical).
10. constant
See para.2: According to the standard view, the temperature of the sun’s core is held constant by the opposing pressures of gravity and nuclear fusion.
11. orbit
See para.6: Most scientists believe that the ice ages are the result of subtle changes in Earth’s orbit, 匛arth’s orbit gradually changes shape from a circle to a slight ellipse and back again roughly every 100,000 years.
12. instabilities
See para.3: ?i style=’magnetic fields in the sun’s core could produce small instabilities in the solar plasma. These instabilities would induce localised oscillations in temperature.
13. cycles
See para.4: …allow the sun’s core temperature to oscillate around its average temperature of 13.6 million kelvin in cycles lasting either 100,000 or 41,000 years.
14. random
See para.4: Ehrlich says that random interactions within the sun’s magnetic field could flip the fluctuations from one cycle length to the other
雅思考试阅读理解提分训练试题及答案
1. The failure of a high-profile cholesterol drug has thrown a spotlight on the complicated machinery that regulates cholesterol levels. But many researchers remain confident that drugs to boost levels of 'good' cholesterol are still one of the most promising means to combat spiralling heart disease.
2. Drug company Pfizer announced on 2 December that it was cancelling all clinical trials of torcetrapib, a drug designed to raise heart-protective high-density lipoproteins (HDLs)。 In a trial of 15000 patients, a safety board found that more people died or suffered cardiovascular problems after taking the drug plus a cholesterol-lowering statin than those in a control group who took the statin alone.
3. The news came as a kick in the teeth to many cardiologists because earlier tests in animals and people suggested it would lower rates of cardiovascular disease. "There have been no red flags to my knowledge," says John Chapman, a specialist in lipoproteins and atherosclerosis at the National Institute for Health and Medical Research (INSERM) in Paris who has also studied torcetrapib. "This cancellation came as a complete shock."
4. Torcetrapib is one of the most advanced of a new breed of drugs designed to raise levels of HDLs, which ferry cholesterol out of artery-clogging plaques to the liver for removal from the body. Specifically, torcetrapib blocks a protein called cholesterol ester transfer protein (CETP), which normally transfers the cholesterol from high-density lipoproteins to low density, plaque-promoting ones. Statins, in contrast, mainly work by lowering the 'bad' low-density lipoproteins.
Under pressure
5. Researchers are now trying to work out why and how the drug backfired, something that will not become clear until the clinical details are released by Pfizer. One hint lies in evidence from earlier trials that it slightly raises blood pressure in some patients. It was thought that this mild problem would be offset by the heart benefits of the drug. But it is possible that it actually proved fatal in some patients who already suffered high blood pressure. If blood pressure is the explanation, it would actually be good news for drug developers because it suggests that the problems are specific to this compound. Other prototype drugs that are being developed to block CETP work in a slightly different way and might not suffer the same downfall.
6. But it is also possible that the whole idea of blocking CETP is flawed, says Moti Kashyap, who directs atherosclerosis research at the VA Medical Center in Long Beach, California. When HDLs excrete cholesterol in the liver, they actually rely on LDLs for part of this process. So inhibiting CETP, which prevents the transfer of cholesterol from HDL to LDL, might actually cause an abnormal and irreversible accumulation of cholesterol in the body. "You're blocking a physiologic mechanism to eliminate cholesterol and effectively constipating the pathway," says Kashyap.
Going up
7. Most researchers remain confident that elevating high density lipoproteins levels by one means or another is one of the best routes for helping heart disease patients. But HDLs are complex and not entirely understood. One approved drug, called niacin, is known to both raise HDL and reduce cardiovascular risk but also causes an unpleasant sensation of heat and tingling. Researchers are exploring whether they can bypass this side effect and whether niacin can lower disease risk more than statins alone. Scientists are also working on several other means to bump up high-density lipoproteins by, for example, introducing synthetic HDLs. "The only thing we know is dead in the water is torcetrapib, not the whole idea of raising HDL," says Michael Miller, director of preventive cardiology at the University of Maryland Medical Center, Baltimore.
Questions 1-7
This passage has 7 paragraphs 1-7.
Choose the correct heading for each paragraph from the list of headings below.
Write the correct number i-ix in boxes 1-7 on your answer sheet.
List of Headings
i. How does torcetrapib work?
ii. Contradictory result prior to the current trial
iii. One failure may possibly bring about future success
iv. The failure doesn't lead to total loss of confidence
v. It is the right route to follow
vi. Why it's stopped
vii. They may combine and theoretically produce ideal result
viii. What's wrong with the drug
ix. It might be wrong at the first place
Questions 7-13
Match torcetrapib,HDLs,statin and CETP with their functions (Questions 8-13)。。
Write the correct letter A, B, C or D in boxes 8-13 on your answer sheet.
NB You may use any letter more than once.
7.It has been administered to over 10,000 subjects in a clinical trial.
8.It could help rid human body of cholesterol.
9.Researchers are yet to find more about it.
10. It was used to reduce the level of cholesterol.
11. According to Kashyap, it might lead to unwanted result if it's blocked.
12. It produced contradictory results in different trials.
13. It could inhibit LDLs.
List of choices
A. Torcetrapic
B. HDLS
C. Statin
D. CETP
Suggested Answers and Explanations
1. vi
2. ii
3. vii 本段介绍了torcetrapib和statin的治病原理,但是同时短语"in contrast"与之前第二段后半段的内容呼应,暗示了这两种药在理论上能相辅相成,是理想的搭配。第一个选项无法涵盖整段意义,故选择i是错误的。
4. iii 本段分析了可能导致torcetrapibl临床试验失败的原因,后半段指出如果以上推测正确,那么未来的药物可借鉴这个试验,设法避免torcetrapib的缺陷,研制出有效的药物。viii选项无法涵盖后半段的意思。
5. ix 见首句。
6. v
7. A 见第二段。题目中administer一词意为"用药",subject一词为"实验对象"之意。
8. B 见第四段"… to raise levels of HDLs, which ferry cholesterol out of artery- clogging plaques to the liver for removal from the body."即HDLs的作用最终是将 choleserol清除出人体:"… for removal from the body"。
9. B 见第四段"But HDLs are complex and not entirely understood."
10. C 见第二段"… plus a cholesterol-lowering statin",即statin是可以降低cholesterol的。
11. D 见第六段 "So inhibiting CETP, … might actually cause an abnormal and irreversible accumulation of cholesterol in the body.
12. A 见第三段。
13. C 见第四段"Statins, in contrast, mainly work by lowering the 'bad' low-density lipoproteins
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